Kuyini CL? Izimpawu zesifo kanye nokwelashwa kwayo.

State ngenxa ukubukeka amatshe e-gallbladder futhi ipheshana biliary, ngokuthi cholelithiasis, cholelithiasis, noma indlela efingqiwe yokubhala igama. Izimpawu zalesi sifo, ikakhulukazi liphinyiselwe ngesikhathi exacerbation. Lesi isifo lalivamile wokugaya ukudla. Ngo yobudala emaphakathi - ngobudala 40, cholelithiasis, izimpawu zalesi sifo singabonakala kula-30% abesifazane 15% amadoda. Kaningi, lapho amatshe atholakala enyongweni wathola ukuvuvukala kwesinye (cishe 35% amacala). Ukubukeka amatshe ahlotshaniswa njalo kahle phakathi Bile acid, lecithin ngakolunye uhlangothi kanti izinto ziyoncibilika Bile (i-cholesterol, bilirubin, i-calcium carbonate) nge Ngakolunye uhlangothi. Ukuze ukulahlekelwa cholesterol inzika, ngempela ekushintshashintsheni awuwona yini ongelutho kunayo lokhu ukulinganisela. Umphumela crystallization Bile futhi fallout yayo inzika. Izimbangela ngokweqile cholesterol eningi. Lena, okokuqala, umkhiqizo wawanda cholesterol, ukunciphisa ukwakheka Bile acid kanye phospholipids. Okwesibili, le mucosa we gallbladder inike glycoproteins ezingakubangela ukugcwaliswa ngokweqile Bile ne-cholesterol. Enye ingxenye yesigaba sokubhekela izingxenye Bile - bilirubin, kunedwala eliyinqaba eliqoshwe cholesterol esitheni, njengoba ukuqinisa ke nekucinisa itshe. I-calculus okuholela kunemiphumela sokugembula: ukwehliselwa itshe esinyeni angamuvimba mafutha anciphisa isikhala esingaphakathi we imbobo, ngakolunye uhlangothi, njalo kuyacasula kwalimala odongeni isinye, kuholela ukuthuthukiswa cholecystitis.

Ukuxilongwa isifo gallstone, izimpawu

izikhalazo esivamile isiguli, ukuthola amatshe esinyeni, njengoba umthetho, ukuvumela kalula ukubeka ukuxilongwa udokotela cholelithiasis. Izimpawu futhi emtholampilo isifo libhekene ukubukeka ngesikhathi exacerbation colic (ezinzima paroxysmal ubuhlungu) e projection kwesinye kanye esibindini. Ubuhlungu kungase nasentanyeni, ngakwesokunene ngaphansi isiphanga. Konke lokhu kungenziwa ephelezelwa isicanucanu, ukunambitheka ababayo emlonyeni nokuhlanza nge Bile. Phakathi ubuhlungu eside, ukuvaleka itshe Amapayipi excretory livela silume futhi angagcina jaundice kwesikhumba futhi sclera. colic Sinzima kungase kuhambisane nezimpawu yokucasuka we peritoneum.

Ngaso sonke isikhathi kufanele bazi kahle ukuthi yini zonke ulahlwa noma exacerbation izifo, ozobakhona amahora ambalwa, uma usuthathe izinyathelo olandelanayo ayizwakali, inkomba esibhedlela emnyangweni kuhlinzwa ukunquma okwengeziwe isu ukwelashwa.

Lapho kwenziwa ucwaningo lwembulunga, kungenzeka ukuthola by palpation futhi kubuhlungu gallbladder lakhulisiwe. Isibindi nakho kungaba kancane lakhulisiwe futhi kabuhlungu. Akhayo ejwayelekile gallstone izimpawu (Ortner, Murphy, Kerala, Zakharyin nabanye).

Njengoba GSD landile abesifazane, kubalulekile ukuba bahlale ngokwehlukana kule nguqulo njengoba CL nokukhulelwa. Uma owesifazane eke waba ngaphambi kokukhulelwa amatshe, noma kukhona nezici ezithile ukuze kumiswa yabo (obukhulu Bile futhi ukusonteka egoba esinyeni, dyskinesia biliary), i-ukukhulelwa kungabangela okubi kakhulu lesi sifo. Kwesibeletho lakhulisiwe ngazo bubble, senze yokujama tsi of Bile futhi crystallization yayo. Ukugcina lezo iziguli exacerbation kunciphisa kungenzeka cupping olandelanayo kanye nokusebenza okwalandela ngemuva kokubeletha. Uma lokhu kuhlasela akuhlobene wayeka, ngokushesha enze cholecystectomy ne imbobo amanzi.

yezinkinga cholelithiasis

Izinkinga lesi sifo kungase kube ukuthuthukiswa cholecystitis ngenxa yokulimala esinyeni odongeni nokunamathela ukutheleleka. Choledoch itshe impaction noma ezinye imbobo hepatic kubangela ukuvaleka (choledocholithiasis), kanye mechanical uhlobo jaundice. Lokhu-ke okubangela ukuvuvukala we mapayipi. I inkinga olunzima kunalo lonke ake isifo gallstone ukuthuthukiswa Bile peritonitis. usizo Ibambezelekile kungaholela ekufeni.

ukwelashwa

ukwelashwa kuhlinzwa CL. Khiqiza bubble ukususwa. Uma amatshe basesimweni ezivamile Bile imbobo, uyivule, concrements sikhishiwe ngemva sutured akhanywa.